renal sclerosis
英 [ˈriːnl skləˈrəʊsɪs]
美 [ˈriːnl skləˈroʊsɪs]
网络 肾硬化
双语例句
- The Treatment of Renal Cysts with Imaging-guided Percutaneous Catheterization Drainage and Alcohol Sclerosis
经皮穿刺置管引流并硬化治疗肾囊肿 - Effect of ligation of renal lymphatic vessels and renal pelvis infusion sclerosis on recurrent severe chyluria: 46 cases observation
肾蒂淋巴管结扎术和RPIS治疗术后复发性重症乳糜尿的疗效 - The degree of the renal parenchyma thickness becoming thin showed renal arteriole sclerosis and the decreased number of remnant nephron.
肾实质变薄的程度表明肾小动脉硬化，残存肾单位总数下降。 - Renal cell carcinoma coexisting with angiomyolipoma in tuberous sclerosis is extremely rare.
肾细胞癌与血管肌肉脂肪瘤同时并存于结节性硬化症病人身上的情况非常少见。 - Effect of prostaglandin E_1 on renal expression of HGF in glomerular sclerosis in rats
前列腺素E1对肾小球硬化大鼠肾组织肝细胞生长因子的调节 - Expressions of metalloproteinase-9 and transform growth factor-β_1 mRNA in peripheral blood monocytes in patients with hypertention and renal arteriolar sclerosis
高血压肾小动脉硬化病人外周血单个核细胞中MMP-9及TGF-β1mRNA的表达 - Liver and renal cyst treated with improved sclerosis therapy
肝肾囊肿硬化疗法的改良 - Conclusion: Fluvastatin can down-regulate the activity of signal transduction pathway-ERK1/ 2, inhibit the overproduction of TGF-β 1, decrease the expression of type ⅳ collagen, improve the renal pathological changes, inhibit and delay the glomerular sclerosis.
结论：氟伐他汀能下调糖尿病大鼠肾皮质ERK1/2活性、抑制TGF-β1过多生成，降低肾皮质Ⅳ型胶原表达，减轻肾脏病理学改变，抑制和延缓肾小球硬化。 - Renal hamartoma and cerebral tuberous sclerosis: two cases report
肾脏错构瘤伴脑结节性硬化2例 - Conclusion: Fluvastatin not only reduces proteinuria, improves renal function, but also modulates glomerular sclerosis by inhibiting activities of TGF-β 1, PAI-1 and decreasing accumulation of collagen type ⅳ. The mechanism of renal protective effect is independent of a reduction of circulating cholesterol.
结论：氟伐他汀可抑制TGF鄄β1及PAI鄄1，减少Ⅳ型胶原积聚，减轻肾小球硬化，降低蛋白尿，改善肾功能，其对肾脏的保护作用并不依赖降胆固醇作用。 - Liver and renal cysts treated with improved sclerosis therapy in the old people
老年人肝肾囊肿硬化疗法的改良&附两种方法的疗效比较 - Clinicopathological studies of renal involvement in patients with systemic sclerosis
系统性硬化症的肾脏损害 - Analysis of efficiency in therapy of ultrasound-guided renal cyst puncture for suction and anhydrous alcohol sclerosis
B超引导下肾囊肿穿刺抽吸及酒精硬化疗效分析 - Renal morphological study revealed that major glomeruli were focal segmental sclerosis. There were a massive protein casts in the dilated tubules and amount of inflammatory cells in interstitium in high cholesterol group.
形态学检查发现，高胆固醇组大部分肾小球局灶节段硬化，小管扩张，可见蛋白管型和间质炎症细胞浸润； - Nowadays, studies on renal protection mainly focus on prevention and treatment of glomerular sclerosis, renal interstitial fibrosis and intrarenal vascular sclerosis etc.
目前，肾保护的研究主要从防治肾小球硬化、肾间质纤维化和肾内血管硬化等方面进行，其中减少细胞外基质（ECM）过度积聚是其核心环节。 - Results: Seventy-two ( 48.3%) of patients have positive staining of amylin in the renal tissue. Amylin was found mainly in the expanded mesangial area, segmental sclerosis, K-W nodules, the Bowman's capsule as well as in blood vessel.
结果72例DN患者（48.3%）肾组织Amylin染色阳性，Amylin主要沉积在肾小球系膜基质增多而扩张的系膜区、K-W结节内、增厚的球囊壁和病变的血管壁。 - Background and Objective: Almost all forms of kidney diseases progressing to end-stage renal failure are characterized by diffuse glomerular sclerosis and interstital fibrosis.
背景与目的：各种慢性肾脏病进展至慢性肾衰的病理改变均以弥漫性肾小球硬化和间质纤维化为特征。 - Pathological examination confirmed that the increase of RRI is related to renal biopsy, vascular wall thickening and glomerular sclerosis.
肾组织穿刺病理检查证实，RRI的升高与肾小血管壁增厚，肾小球退行性变或硬化改变有关。 - Recent studies demonstrated that miRNA could regulate EMT in different kind of cells and it was shown effective to inhibit organ fibrosis, such as cardiac, liver, lung, renal fibrosis as well as systemic sclerosis, by targeting miRNAs to reverse EMT.
最新的研究表明，miRNAs参与调节多种组织细胞EMT过程，靶向miRNA可以有效逆转EMT并抑制器官纤维化，包括心脏、肝脏、肺、肾纤维化，系统性硬化等。 - At 8w post-injection, the renal size had a decreasing trend and pathological studies showed glomerular sclerosis, renal interstitial fibrosis and tubular atrophy.
8w时肾脏出现减小的趋势，病理出现肾小球硬化、间质纤维化和肾小管萎缩。 - Its main mechanism maybe concluded as follows: Yishentongluo Decoction can exhibit extracellular matrix ( ECM) proliferation and accumulation in renal tissues of rats with nephrotic syndrome, postpone sclerosis in renal glomerulus, postpone the process of fibrosis in renal interstitial substance to protect renal tissues.
这可能是益肾通络方抑制NS大鼠肾组织细胞外基质（ECM）的增生和积聚，延缓了肾小球硬化和肾间质纤维化的进程保护肾组织的主要机理。 - A large number of studies have shown that renal interstitial fibrosis and renal function of the degree of correlation than the glomerular sclerosis and renal function is more closely related.
大量研究表明，肾间质纤维化的程度与肾功能的相关性比肾小球硬化与肾功能的相关性更为密切。 - The major renal pathological changes of diabetic nephropathy both developed from type 1 and type 2 diabetes are glomerular sclerosis.
肾小球硬化是1型与2型DN共同的特征性肾脏病理改变。 - Severe renal hyperblastosis, glomerulus sclerosis, crescent formation, kidney tubule atrophy, mesenchyma fibrosis, and vessel damages are regarded as indicators of poor prognosis for isolated hematuria.
严重的肾组织增生、肾小球硬化、新月体形成、肾小管萎缩、间质纤维化和血管损害，是孤立性血尿预后不良的标志。 - Background: A common pathway to end stage renal failure ( ESRF) caused by all kinds of kidney diseases is renal interstitial fibrosis and sclerosis, which lead to the loss of effective nephron and the progressive decline of renal function.
背景：不同病因引起的慢性肾脏疾病发展为终末期肾衰竭（endstagerenalfailure，ESRF）的共同途径是肾间质纤维化和硬化，最终导致有效肾单位的丧失和肾功能的进行性下降。 - The main pathological alteration is the accumulation of extracellular matrix aggregation on renal tubular and renal glomerulus. Accordingly, it can result in glomerular sclerosis and renal interstitial fibrosis.
其主要的病理改变是细胞外基质（ECM）在肾小球、肾小管聚集，最终导致肾小球硬化及肾小管间质纤维化。